Wednesday, August 26, 2009

JAPALA


Small shrub or tree up to 12 m tall, evergreen; leaves alternate, membranous, ovate with broadly rounded, sometimes slightly decurrent base, acuminate, acute or blunt, very shallowly serrate, glabrous above, with few stellate hairs beneath, 7.5-17 cm long, 4-9.5 cm broad, metallic green to bronze or orange; petiole slender, about 4 cm long; stipules caducous, subulate, 1.5- 3.5 mm long; axis of inflorescence glabrous; flowers small, inconspicuous; male flowers stellately hairy with narrowly oblong petals and 15-20 stamens; female flowers apetalous; capsule scabrid with stellate hairs, triangular, 15-20 mm long, 10-15 mm broad, oblong or ellipsoid, 3-lobed; seeds 3 per fruit, oblong-ovoid, orange, about 12 mm long, C.S.I.R. reports that the oil contains 3.4% toxic resin. Of the acids, 37.0% is oleic, 19.0% linoleic, 1.5% arachidic, 0.3% stearic, 0.9% palmitic, 7.5% myristic, 0.6% acetic, 0.8% formic, with traces of lauric, tiglic, valeric, and butyric, plus some unidentified.
to New Guinea and Java, north into Indonesia and China. Wild throughout the Philippine Islands, where it is also cultivated to a limited extent; often becoming naturalized after cultivation. Grown in southern California and elsewhere as an ornamental and curious plant.

WHAT IS POISONUS PLANT

Poisonous Plants, plants containing substances that, taken into the body of humans or animals in small or moderate amounts, provoke a harmful reaction resulting in illness or death. Possibly as many as one out of each 100 species of plants is poisonous, but not all have been recognized as such. Dangerous plants are widely distributed in woods (baneberry) and fields (star-of-Bethlehem), swamps (false hellebore) and dry ranges (scrub oak), roadsides (climbing bittersweet) and parks (kalmia), and may be wild (celandine) or cultivated (wisteria). Many ornamental plants, such as oleander, lily of the valley, and mistletoe, are poisonous.
Botanists have no set rules to determine accurately whether any given plant is poisonous. Toxic species are scattered geographically, in habitat, and in botanical relationship. They contain more than 20 kinds of poisonous principles, primarily alkaloids, glycosides, saponins, resinoids, oxalates, photosensitizing compounds, and mineral compounds such as selenium or nitrates accumulated from the soil. The poisonous compound may be distributed throughout all parts of the plant (poison hemlock), or it may accumulate in one part more than any other, such as the root (water hemlock), berry (daphne), or foliage (wild cherries). A plant may vary in toxicity as it grows, generally becoming more toxic with maturity; certain plants, however, can be highly toxic when young and harmless later (cocklebur).
Some active principles cause skin irritation directly (nettle); others bring about an allergenic reaction (poison ivy). Most poisons, however, must enter the body before they act, and in almost all cases this happens when they are eaten. Usually more than 57 g (2 oz) of the poisonous portion of the plant must be eaten by an average adult before poisoning will result (the amount is proportionately less for children). Some plants, however, are toxic in small amounts; for instance, one or two castor beans, the seeds of the castor-oil plant, may kill a child.
After ingestion, the poison may act immediately on the digestive tract (dumbcane, euphorbia, nightshade), producing severe abdominal pain, vomiting, and possibly internal bleeding, or it may be absorbed into the bloodstream. If so, it passes first to the liver, which may be injured. Oxalates crystallize in the kidneys (rhubarb), rupturing the tubules. Some plants affect the heart (oleander). Small amounts of principles in some of these (digitalis) may be used in medicine. Plants containing alkaloids often produce unpleasant or dangerous reactions in the nervous system. Examples are paralysis (poison hemlock), hallucinations (jimsonweed), or heart block (yew).
A few poisons act directly within the cells of the body. The best example is cyanide, released from a glycoside in the plant (wild cherries), which prevents cells of the body from using oxygen. In contrast, unusually high levels of nitrates in plants combine with the hemoglobin of the blood so that it can no longer carry oxygen to the body cells. Some reactions are highly specific. Bracken destroys bone marrow, in which blood cells are formed. Saint John’s wort contains a poison that, when ingested by animals, reacts with sunlight to produce severe sunburn and lesions on exposed skin.
Poisonous plants are too numerous to eradicate, and many are highly prized as houseplants or garden ornamentals. If poisoning is suspected, a physician or the local poison control center should be consulted immediately. See also Poison.

GODA KADURU




Strychnos Tree
The strychnos tree yields a varied crop of poison, medicine, and food. Aboriginal groups use the bark and leaves of the plant to make curare, a paralyzing poison used in hunting. Some species of the plant yield a quininelike medicine; and the fruit of the natal orange variety is cultivated for commercial use.

Caster been (Edaru)


The seeds from the castor bean plant, Ricinus communis, are poisonous to people, animals and insects. One of the main toxic proteins is "ricin", named by Stillmark in 1888 when he tested the beans' extract on red blood cells and saw them agglutinate. Now we know that the agglutination was due to another toxin that was also present, called RCA (Ricinus communis agglutinin). Ricin is a potent cytotoxin but a weak hemagglutinin, whereas RCA is a weak cytotoxin and a powerful hemagglutinin.
Poisoning by ingestion of the castor bean is due to ricin, not RCA, because RCA does not penetrate the intestinal wall, and does not affect red blood cells unless given intravenously. If RCA is injected into the blood, it will cause the red blood cells to agglutinate and burst by hemolysis.
Perhaps just one milligram of ricin can kill an adult.
The symptoms of human poisoning begin within a few hours of ingestion.
The symptoms are:
abdominal pain
vomiting
diarrhea, sometimes bloody.
Within several days there is:
severe dehydration,
a decrease in urine,
and a decrease in blood pressure.
If death has not occurred in 3-5 days, the victim usually recovers. It is advisable to keep children away from the castor bean plant or necklaces made with its seeds. In fact donĂ­t even have them in or around a house with small children. If they ingest the leaves or swallow the seeds, they may get poisoned. The highly toxic seeds beaded into necklaces, cause skin irritation at the contact point.
If the seed is swallowed without chewing, and there is no damage to the seed coat, it will most likely pass harmlessly through the digestive tract. However, if it is chewed or broken and then swallowed, the ricin toxin will be absorbed by the intestines.
It is said that just one seed can kill a child. Children are more sensitive than adults to fluid loss due to vomiting and diarrhea, and can quickly become severely dehydrated and die.
Castor bean plants in a garden should not be allowed to flower and seed.

Tuesday, August 25, 2009


Thevetia peruviana (pers). K Shum1.2

FamilyApocynaceae1.3 Common name(s)Be still TreeDigoxinLucky NutNerium oleanderYellow Oleander2.

SUMMARY2.1 Main risks and target organsThe main toxic effects of glycosides found in Thevetiaperuviana are related to its digitalis-like action on theheart and severe gastrointestinal irritation.

2.2 Summary of clinical effectsThe common presenting symptoms are: numbness, burning of themouth, nausea, vomiting, abdominal pain and diarrhoea. Otherfeatures seen are: drowsiness, coma, occasional convulsions,and cardiac arrhythmias. Death is due to ventricularfibrillation.

2.3 DiagnosisDiagnosis depends on the patient's history and the clinicalpresentation. Cardiac glycosides can be investigated in the blood bycompetitive immunoassay. The serum potassium concentrationshould be monitored. Electrocardiography, frequent serumelectrolytes (especially potassium concentration), and renalfunction tests are essential investigations. Remnants ofseeds, vomitus or gastric aspirate should be collected foridentification purposes.

2.4 First-aid measures and management principlesAdmit the patient to a hospital. Treatment should aim at a)gut contamination by emesis or lavage if the ingestion isrecent; b) correction of electrolyte imbalance; c) correctionof severe bradycardia with atropine or electrical pacing andcorrection of ventricular dysrrhythmias; and d)administration of digoxin Fab antibodies if available.

2.5 Poisonous partsAll parts of the plant, particularly the seeds are poisonousowing to the presence of cardiac glycosides or cardiac toxinswhich act directly on the heart. Ingestion of these plantparts could lead to death. The whole plant exudes in a milkyjuice which is very poisonous.

2.6 Main toxinsThevetin A, Thevetin B and Peruvoside.

3. CHARACTERISTICS3.1 Description of the plant

3.1.1 Special identification featuresIt is a small ornamental tree which grows to about 10 to15 feet high. The leaves are spirally arranged, linearand about 13 to 15 cm in length. Flowers are brightyellow and funnel-shaped with 5 petals spirally twisted. The fruits are somewhat globular, slightlyfleshy and have a diameter of 4 to 5 cm. The fruits,which are green in colour, become black on ripening.Each fruit contains a nut which is longitudinally andtransversely divided. All parts of the plant containthe milky juice.

3.1.2 HabitatGrown as an ornamental tree in gardens.

3.1.3 DistributionThis plant is native of Central & South America, but nowfrequently grown throughout the tropical and sub-tropical regions.

3.2 Poisonous parts of the plantAll parts of the plant are poisonous, especially the kernelsof the fruit. The absorption of the equivalent of two Thevetia peruvianaleaves may be sufficient to kill a 12.5 kg child (Ellenhornand Barceloux, 1988).

CACTAS

Family
Cactaceae
Plant Description
Succulent, flattened stems or shrublike with cylindric stems; leaves lacking or modified into large spines; sharp-pointed glochids in clusters at base of spines; flowers variously colored, with many petals; fruit fleshy or dry.
Origin
Americas.
Distribution
Coastal Plain, Piedmont.
Where Found
Houseplant or interiorscape; Landscape as cultivated ornamental plants, native and naturalized, weedy in disturbed areas, lawns in coastal areas.
Mode
Ingestion; splinter-like small infection due to glochids lodged in the skin, eye irritation.
Poisonous Part
Glochids (minute bristle-like, barbed hairs in clusters) on the stems (green, thickened stems resemble leaves).
Symptoms
Painful skin and eye irritation following contact; internal effects in diabetics from ingestion.
Edibility
EDIBLE PARTS: Ripe fruit edible raw or in jelly. Leaf pads, fruit and seeds. HARVEST TIME: Only collect plants from areas you know have NOT been treated with pesticides. Use tender young leaf pads gathered during the spring. SAFE HANDLING PROCEDURES: Wash leaf pads, fruit and seeds thoroughly with warm water. Do not use dish detergent or any type of sanitizer. These products can leave a residue. Peel and cut pulp into chunks or strips and cook like string beans. Batter, roast or fry pads. The interior of the pad similar to okra and can be used to thicken soups. Cut pads into pieces and use raw in salads. Remove bristles before use with a flame or by wiping off with a glove or damp cloth. Or, bake the pads in a medium-temperature oven for one-half hour, then peel the skin with the bristles attached. If a knife is used to cut out bristles, wipe after each cut, because mucilage produced by the pads will stick to blade. Roast the pads in their skin on a fire for about 15-20 minutes per side. Peel and eat the pulp after cooking. Peel or cut in half and scoop out pulp before use. Chill and eat raw or pickle after removing seeds. Dried seeds can be crushed or ground into flour and used in soup as a thickener. SOURCE: Larson, Ken. 1995. God's Free Harvest, Rhema Publishing, Inc., Suwanee, GA. 231 pp.
Toxic Principle
Unknown; possibly mechanical effect of glochids.
Severity
CAUSES ONLY LOW TOXICITY IF EATEN. SKIN IRRITATION SEVERE!

MANIOC

Family
Euphorbiaceae
Plant Description
A bushy herb or shrub with elongated tubers; leaves alternate, simple, deeply lobed; flowers inconspicuous; fruit a 6-angled globose capsule.
Origin
Brazil.
Where Found
Plant not grown in North Carolina, imported, purchased in grocery stores, widely cultivated in the tropics for the tuberous roots.
Mode
Ingestion.
Poisonous Part
Raw roots or peelings of the tubers (thickened roots).
Symptoms
Weak and irregular breathing, weakness, excitement, depression, staggering, pupil dilation, spasms, convulsions, coma of short duration.
Edibility
EDIBLE PARTS: Tubers boiled and eaten or made into tapioca.
Toxic Principle
Cyanogenic glycosides linamarin and lotaustralin.
Severity
HIGHLY TOXIC, MAY BE FATAL IF EATEN!